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Difference between hypovolemic shock and heart arrest caused by hypovolemia?

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Is there a difference between hypovolemic shock and heart arrest caused by hypovolemia ?

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5

Is there a difference between hypovolemic shock and heart arrest caused by hypovolemia ?

heart

share|improve this question

asked yesterday

AtanasAtanas

774

• 
  
  
  

  
  1
  

  
  
  
  
  
  

  
  
  What research have you done? Simply looking up the meaning of these terms would answer the question.
  
  – Carey Gregory♦
  yesterday
  

  
  
  
  


• 
  
  
  

  
  1
  

  
  
  
  
  
  

  
  
  Please read the help center. For reasons mentioned in this post and in How to Ask, we require some degree of prior research when asking questions. I'm not going to close this question because it has a good answer with upvotes, but future questions without prior research may be closed.
  
  – Carey Gregory♦
  yesterday
  
  
  

  
  
  
  

add a comment | 

Is there a difference between hypovolemic shock and heart arrest caused by hypovolemia ?

heart

share|improve this question

asked yesterday

AtanasAtanas

774

share|improve this question

asked yesterday

AtanasAtanas

774

share|improve this question

asked yesterday

AtanasAtanas

774

asked yesterday

AtanasAtanas

774

asked yesterday

AtanasAtanas

774

1 Answer
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7

Hypovolemic shock may be a reversible cause of cardiac arrest (5)

Mechanisms:

• Acute cardiac ischemia and myocardial infarction: these are due to decreased oxygen supply to the heart itself (remember that shock itself represent global ischemia)(1)(2)




• pH changes, especially acidemia: when shock reaches to a irreversible state so much acidosis has developed(1) which itself can cause a cardiac arrest.(2)




• Electrolyte abnormalities, most notably hypokalemia, hyperkalemia, and hypomagnesemia: ischemia (untreated) leads infarction leads necrosis leads loss of cell membrane leads ions and enzymes to leak and and produce toxic effects.(2) (3)




• Heart Failure:The incidence of sudden cardiac death appears to be increased during periods of worsening HF symptoms.4.

While Hypovolemic Shock itself does not involve cardiac arrest,

• if uncompensated will ultimately cause tissue damage, release of destructive enzymes, acidosis, depletion of cellular ATP. (1)

Pathophysiology:

As volume status continues to decrease, systolic blood pressure drops. As a result, oxygen delivery to vital organs is unable to meet oxygen demand. Cells switch from aerobic metabolism to anaerobic metabolism, resulting in lactic acidosis.

As sympathetic drive increases, blood flow is diverted from other organs to preserve blood flow to the heart and brain.

This propagates tissue ischemia and worsens lactic acidosis. If not corrected, there will be worsening hemodynamic compromise and, eventually, death.(4)

References:

1. Guyton and Hall Medical Physiology




2. https://www.uptodate.com/contents/pathophysiology-and-etiology-of-sudden-cardiac-arrest#H1240264379




3. Robbins and Cotran Pathologic Basis of Diseases




4. https://www.ncbi.nlm.nih.gov/books/NBK513297/




5. https://en.m.wikipedia.org/wiki/Hs_and_Ts

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edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

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7

Hypovolemic shock may be a reversible cause of cardiac arrest (5)

Mechanisms:

• Acute cardiac ischemia and myocardial infarction: these are due to decreased oxygen supply to the heart itself (remember that shock itself represent global ischemia)(1)(2)




• pH changes, especially acidemia: when shock reaches to a irreversible state so much acidosis has developed(1) which itself can cause a cardiac arrest.(2)




• Electrolyte abnormalities, most notably hypokalemia, hyperkalemia, and hypomagnesemia: ischemia (untreated) leads infarction leads necrosis leads loss of cell membrane leads ions and enzymes to leak and and produce toxic effects.(2) (3)




• Heart Failure:The incidence of sudden cardiac death appears to be increased during periods of worsening HF symptoms.4.

While Hypovolemic Shock itself does not involve cardiac arrest,

• if uncompensated will ultimately cause tissue damage, release of destructive enzymes, acidosis, depletion of cellular ATP. (1)

Pathophysiology:

As volume status continues to decrease, systolic blood pressure drops. As a result, oxygen delivery to vital organs is unable to meet oxygen demand. Cells switch from aerobic metabolism to anaerobic metabolism, resulting in lactic acidosis.

As sympathetic drive increases, blood flow is diverted from other organs to preserve blood flow to the heart and brain.

This propagates tissue ischemia and worsens lactic acidosis. If not corrected, there will be worsening hemodynamic compromise and, eventually, death.(4)

References:

1. Guyton and Hall Medical Physiology




2. https://www.uptodate.com/contents/pathophysiology-and-etiology-of-sudden-cardiac-arrest#H1240264379




3. Robbins and Cotran Pathologic Basis of Diseases




4. https://www.ncbi.nlm.nih.gov/books/NBK513297/




5. https://en.m.wikipedia.org/wiki/Hs_and_Ts

share|improve this answer

edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

3796

add a comment | 

7

Hypovolemic shock may be a reversible cause of cardiac arrest (5)

Mechanisms:

• Acute cardiac ischemia and myocardial infarction: these are due to decreased oxygen supply to the heart itself (remember that shock itself represent global ischemia)(1)(2)




• pH changes, especially acidemia: when shock reaches to a irreversible state so much acidosis has developed(1) which itself can cause a cardiac arrest.(2)




• Electrolyte abnormalities, most notably hypokalemia, hyperkalemia, and hypomagnesemia: ischemia (untreated) leads infarction leads necrosis leads loss of cell membrane leads ions and enzymes to leak and and produce toxic effects.(2) (3)




• Heart Failure:The incidence of sudden cardiac death appears to be increased during periods of worsening HF symptoms.4.

While Hypovolemic Shock itself does not involve cardiac arrest,

• if uncompensated will ultimately cause tissue damage, release of destructive enzymes, acidosis, depletion of cellular ATP. (1)

Pathophysiology:

As volume status continues to decrease, systolic blood pressure drops. As a result, oxygen delivery to vital organs is unable to meet oxygen demand. Cells switch from aerobic metabolism to anaerobic metabolism, resulting in lactic acidosis.

As sympathetic drive increases, blood flow is diverted from other organs to preserve blood flow to the heart and brain.

This propagates tissue ischemia and worsens lactic acidosis. If not corrected, there will be worsening hemodynamic compromise and, eventually, death.(4)

References:

1. Guyton and Hall Medical Physiology




2. https://www.uptodate.com/contents/pathophysiology-and-etiology-of-sudden-cardiac-arrest#H1240264379




3. Robbins and Cotran Pathologic Basis of Diseases




4. https://www.ncbi.nlm.nih.gov/books/NBK513297/




5. https://en.m.wikipedia.org/wiki/Hs_and_Ts

share|improve this answer

edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

3796

add a comment | 

Hypovolemic shock may be a reversible cause of cardiac arrest (5)

Mechanisms:

• Acute cardiac ischemia and myocardial infarction: these are due to decreased oxygen supply to the heart itself (remember that shock itself represent global ischemia)(1)(2)




• pH changes, especially acidemia: when shock reaches to a irreversible state so much acidosis has developed(1) which itself can cause a cardiac arrest.(2)




• Electrolyte abnormalities, most notably hypokalemia, hyperkalemia, and hypomagnesemia: ischemia (untreated) leads infarction leads necrosis leads loss of cell membrane leads ions and enzymes to leak and and produce toxic effects.(2) (3)




• Heart Failure:The incidence of sudden cardiac death appears to be increased during periods of worsening HF symptoms.4.

While Hypovolemic Shock itself does not involve cardiac arrest,

• if uncompensated will ultimately cause tissue damage, release of destructive enzymes, acidosis, depletion of cellular ATP. (1)

Pathophysiology:

As volume status continues to decrease, systolic blood pressure drops. As a result, oxygen delivery to vital organs is unable to meet oxygen demand. Cells switch from aerobic metabolism to anaerobic metabolism, resulting in lactic acidosis.

As sympathetic drive increases, blood flow is diverted from other organs to preserve blood flow to the heart and brain.

This propagates tissue ischemia and worsens lactic acidosis. If not corrected, there will be worsening hemodynamic compromise and, eventually, death.(4)

References:

1. Guyton and Hall Medical Physiology




2. https://www.uptodate.com/contents/pathophysiology-and-etiology-of-sudden-cardiac-arrest#H1240264379




3. Robbins and Cotran Pathologic Basis of Diseases




4. https://www.ncbi.nlm.nih.gov/books/NBK513297/




5. https://en.m.wikipedia.org/wiki/Hs_and_Ts

share|improve this answer

edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

3796

share|improve this answer

edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

3796

edited 18 hours ago

Carey Gregory♦

7,23831742

edited 18 hours ago

Carey Gregory♦

7,23831742

answered yesterday

PhysicsapprovalPhysicsapproval

3796

answered yesterday

PhysicsapprovalPhysicsapproval

3796